H-KYNKANVFL-OH

Ref. 3D-PP43811

Informations sur le produit

• Nom: H-KYNKANVFL-OH
• Synonymes: NH2-Lys-Tyr-Asn-Lys-Ala-Asn-Val-Phe-Leu-OH

Description

Peptide H-KYNKANVFL-OH is a Research Peptide with significant interest within the field academic and medical research. This peptide is available for purchase at Cymit Quimica in multiple sizes and with a specification of your choice. Recent citations using H-KYNKANVFL-OH include the following: TLR9 blockade inhibits activation of diabetogenic CD8+ T cells and delays autoimmune diabetes Y Zhang, AS Lee, A Shameli , X Geng - The Journal of , 2010 - journals.aai.orghttps://journals.aai.org/jimmunol/article/184/10/5645/38735 Synergistic reversal of type 1 diabetes in NOD mice with anti-CD3 and interleukin-1 blockade: evidence of improved immune regulation V Ablamunits, O Henegariu, JB Hansen - Diabetes, 2012 - Am Diabetes Assochttps://diabetesjournals.org/diabetes/article-abstract/61/1/145/15859 Pre-existing autoimmunity determines type 1 diabetes outcome after Flt3-ligand treatment TL Van Belle , T Juntti, J Liao, MG von Herrath - Journal of autoimmunity, 2010 - Elsevierhttps://www.sciencedirect.com/science/article/pii/S0896841109001504 Requirement for both H-2Db and H-2Kd for the induction of diabetes by the promiscuous CD8+ T cell clonotype AI4 T Takaki, SM Lieberman , TM Holl, B Han - The Journal of , 2004 - journals.aai.orghttps://journals.aai.org/jimmunol/article/173/4/2530/72612 Antibody combination therapy targeting CD25, CD70 and CD8 reduces islet inflammation and improves glycaemia in diabetic mice T Alkhamis, J Barbic - Clinical & , 2012 - academic.oup.comhttps://academic.oup.com/cei/article-abstract/170/2/139/6428987 Individual nonobese diabetic mice exhibit unique patterns of CD8+ T cell reactivity to three islet antigens, including the newly identified widely expressed dystrophia SM Lieberman , T Takaki, B Han - The Journal of , 2004 - journals.aai.orghttps://journals.aai.org/jimmunol/article/173/11/6727/2076 Identification of the beta cell antigen targeted by a prevalent population of pathogenic CD8+ T cells in autoimmune diabetes SM Lieberman , AM Evans, B Han - Proceedings of the , 2003 - National Acad Scienceshttps://www.pnas.org/doi/abs/10.1073/pnas.0932778100 Stimulation of a shorter duration in the state of anergy by an invariant natural killer T cell agonist enhances its efficiency of protection from type 1 diabetes R Tohn, H Blumenfeld, SMM Haeryfar - Clinical & , 2011 - academic.oup.comhttps://academic.oup.com/cei/article-abstract/164/1/26/6428852 Minimal Impact of a De Novo-Expressed beta-Cell Autoantigen on Spontaneous Diabetes Development in NOD Mice MM Martinic, AE Juedes, D Bresson, D Homann - Diabetes, 2007 - Am Diabetes Assochttps://diabetesjournals.org/diabetes/article-abstract/56/4/1059/12999 Insulin-producing intestinal K cells protect nonobese diabetic mice from autoimmune diabetes M Mojibian , AWY Lam, Y Fujita, A Asadi , GA Grassl - Gastroenterology, 2014 - Elsevierhttps://www.sciencedirect.com/science/article/pii/S0016508514003692 NKG2D blockade prevents autoimmune diabetes in NOD mice K Ogasawara, JA Hamerman, LR Ehrlich - Immunity, 2004 - cell.comhttps://www.cell.com/immunity/pdf/S1074-7613(04)00140-2.pdf Limitations of IL-2 and Rapamycin in Immunotherapy of Type 1 Diabetes J Woytschak, O Boyman, A Hartemann, E Piaggio - 2013 - academia.eduhttps://www.academia.edu/download/44971940/Limitations_of_IL-2_and_Rapamycin_in_Imm20160421-19291-1dorixa.pdf Ins2 Deficiency Augments Spontaneous HLA-A* 0201-Restricted T Cell Responses to Insulin I Jarchum, TP DiLorenzo - The journal of immunology, 2010 - journals.aai.orghttps://journals.aai.org/jimmunol/article/184/2/658/111847 Islet lymphocyte subsets in male and female NOD mice are qualitatively similar but quantitatively distinct EF Young, PR Hess, LW Arnold, R Tisch - Autoimmunity, 2009 - Taylor & Francishttps://www.tandfonline.com/doi/abs/10.3109/08916930903213993 Predominant occupation of the class I MHC molecule H-2Kwm7 with a single self-peptide suggests a mechanism for its diabetes-protective effect DR Brims, J Qian, I Jarchum, L Mikesh - International , 2010 - academic.oup.comhttps://academic.oup.com/intimm/article-abstract/22/3/191/792084 Novel leptin receptor mutation in NOD/LtJ mice suppresses type 1 diabetes progression: II. Immunologic analysis CH Lee, YG Chen , J Chen, PC Reifsnyder - Diabetes, 2006 - Am Diabetes Assochttps://diabetesjournals.org/diabetes/article-abstract/55/1/171/12307 Modulation ofT cell antigen receptor signaling in CDS+ T lymphocytes following priming with homeostatic and inflammatory cytokines C Lamontagne-Blouin - 2012 - savoirs.usherbrooke.cahttps://savoirs.usherbrooke.ca/bitstream/handle/11143/6325/Lamontagne_Blouin_Christopher_MSc_2012.pdf?sequence=2 Improved outcomes in NOD mice treated with a novel Th2 cytokine-biasing NKT cell activator C Forestier, T Takaki, A Molano , JS Im - The Journal of , 2007 - journals.aai.orghttps://journals.aai.org/jimmunol/article/178/3/1415/74175 The CD137 ligand is important for type 1 diabetes development but dispensable for the homeostasis of disease-suppressive CD137+ FOXP3+ regulatory CD4 T cells BM Foda, AE Ciecko, DV Serreze - The Journal of , 2020 - journals.aai.orghttps://journals.aai.org/jimmunol/article/204/11/2887/107559 Level of major histocompatibility complex class I expression on endothelium in non-obese diabetic mice influences CD8 T cell adhesion and migration B Lozanoska-Ochser, M Peakman - Clinical & Experimental , 2009 - academic.oup.comhttps://academic.oup.com/cei/article-abstract/157/1/119/6429015 Memory alloreactive B cells and alloantibodies prevent anti-CD154-mediated allograft acceptance AM Burns, L Ma, Y Li, D Yin, JK Shen, J Xu - The Journal of , 2009 - journals.aai.orghttps://journals.aai.org/jimmunol/article/182/3/1314/83659

• Marque: Biosynth