10-493279 - fmoc-l-cysteine-amide

<p>Fmoc-S-4-methoxytrityl-L-cysteine is a synthetic, amphipathic, amide, ester linkages, cyclic peptide. It has been shown to be a potent growth factor that inhibits the proliferation of human pancreatic cancer cells in tissue culture and induces apoptosis in these cells. Fmoc-S-4-methoxytrityl-L-cysteine also inhibits the growth of staphylococcus in tissue culture and reduces metastatic pancreatic cancer cell proliferation in rats.</p>

Fmoc-S-acetamidomethyl-L-cysteine is a peptide analog that has been shown to inhibit the growth of HIV in human prostate cells. It binds to the du-145 cell receptor and inhibits the proteolytic activity of HIV, which may be due to its ability to form an amide bond or disulfide bond with the Cys residue (Cys-X). Fmoc-S-acetamidomethyl-L-cysteine also inhibits the growth of prostate cancer cells by binding to human serum albumin. This drug has minimal toxicity in fetal bovine serum and is not toxic to most bacteria, as it does not bind with glycopeptides, antimicrobial peptides, or proteins. Fmoc-S-acetamidomethyl-L-cysteine has been shown to have biological properties against Trichophyton mentagrophytes and Staphylococcus a

<p>Bachem ID: 4096178.</p>

Fmoc-Cys-NH2

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Fmoc-L-aspartic acid alpha-amide is a prodrug that is activated by hydrolysis in the presence of a cysteine residue. It inhibits protein synthesis by binding to the active site of the enzyme and modifying it, thereby preventing formation of peptide bonds. Fmoc-L-aspartic acid alpha-amide has been shown to be more stable than other aspartic acids because it contains an alpha amide group at its N terminus, which can be cleaved by enzymes and thus easily metabolized. Fmoc-L-aspartic acid alpha-amide is also resistant to proteolytic degradation due to its covalent nature. The modification of the active site allows for selective inhibition of specific proteins involved in inflammation and downregulation of immune responses.