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LCBiot-GRPRTTSFAE-OH
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LCBiot-GRPRTTSFAE-OH

Ref. 3D-PP45574

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Estimated delivery in United States, on Tuesday 3 Dec 2024

Product Information

Name:
LCBiot-GRPRTTSFAE-OH
Synonyms:
  • LCBiot-Gly-Arg-Pro-Arg-Thr-Thr-Ser-Phe-Ala-Glu-OH
Description:

Peptide LCBiot-GRPRTTSFAE-OH is a Research Peptide with significant interest within the field academic and medical research. This peptide is available for purchase at Cymit Quimica in multiple sizes and with a specification of your choice. Recent citations using LCBiot-GRPRTTSFAE-OH include the following: Ezrin-radixin-moesin proteins are regulated by Akt-GSK3beta signaling in the rat nucleus accumbens core WY Kim, WT Cai, JK Jang, JH Kim - The Korean Journal of , 2020 - ncbi.nlm.nih.govhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC6940492/ Unravelling AKT2 signalling in cancer through nanobody technology T Merckaert - 2021 - biblio.ugent.behttps://biblio.ugent.be/publication/8705129/file/8705131 Synthesis of GSK3beta mimetic inhibitors of Akt featuring a novel extended dipeptide surrogate S Ranatunga, JR Del Valle - Bioorganic & medicinal chemistry letters, 2011 - Elsevierhttps://www.sciencedirect.com/science/article/pii/S0960894X11013230 Preparation and Evaluation of Deconstruction Analogs of 7-deoxykalafungin as Akt Inhibitors S Korwar - 2012 - scholarscompass.vcu.eduhttps://scholarscompass.vcu.edu/etd/387/ Hierarchical organization endows the kinase domain with regulatory plasticity P Creixell , JP Pandey, A Palmeri , M Bhattacharyya - Cell systems, 2018 - cell.comhttps://www.cell.com/cell-systems/pdf/S2405-4712(18)30355-7.pdf Akt kinase activation mechanisms revealed using protein semisynthesis N Chu , AL Salguero, AZ Liu , Z Chen , DR Dempsey - Cell, 2018 - cell.comhttps://www.cell.com/cell/pdf/S0092-8674(18)30897-3.pdf Modifications of the GSK3beta substrate sequence to produce substrate-mimetic inhibitors of Akt as potential anti-cancer therapeutics KJ Kayser, MP Glenn, SM Sebti, JQ Cheng - Bioorganic & medicinal , 2007 - Elsevierhttps://www.sciencedirect.com/science/article/pii/S0960894X07000352 An ATP-site on-off switch that restricts phosphatase accessibility of Akt K Lin, J Lin, WI Wu, J Ballard, BB Lee, SL Gloor - Science , 2012 - science.orghttps://www.science.org/doi/abs/10.1126/scisignal.2002618 The regulatory role of leptin in the nucleus accumbens in cocaine-induced locomotor activity and its signaling pathway 읎정원 - 2018 - ir.ymlib.yonsei.ac.krhttps://ir.ymlib.yonsei.ac.kr/bitstream/22282913/160046/1/T014642.pdf Pocketome: an encyclopedia of small-molecule binding sites in 4D I Kufareva , AV Ilatovskiy , R Abagyan - Nucleic acids research, 2012 - academic.oup.comhttps://academic.oup.com/nar/article-abstract/40/D1/D535/2903425 An Update 2009-2012 to"Targeted Small-Molecule Inhibitors of Protein Kinase B as Anticancer Agents" I Collins - Advances in Anticancer Agents in Medicinal Chemistry, 2013 - books.google.comhttps://books.google.com/books?hl=en&lr=&id=Az0VDgAAQBAJ&oi=fnd&pg=PA43&dq=(%22LCBiot-Gly-Arg-Pro-Arg-Thr-Thr-Ser-Phe-Ala-Glu-OH%22+OR+%22LCBiot-GRPRTTSFAE-OH%22+OR+%22GRPRTTSFAE%22)+AND+peptide&ots=LbsjY76p7b&sig=D757sUnfD7uaj8gL98pGo0FH2fI Understanding the relative affinity and specificity of the substrate binding site of protein kinase B for substrate-mimetic inhibitors H Qian, S Chen, Y Pan, J Chen - Molecular Simulation, 2017 - Taylor & Francishttps://www.tandfonline.com/doi/abs/10.1080/08927022.2017.1319062 Glycogen Synthase Kinase 3beta Is a Key Regulator in the Inhibitory Effects of Accumbal Cocaine-and Amphetamine-Regulated Transcript Peptide 55-102 on BR Cho , WY Kim, JK Jang, JW Lee, JH Kim - International Journal of , 2022 - mdpi.comhttps://www.mdpi.com/1422-0067/23/24/15633 Glycogen synthase kinase (GSK) 3beta phosphorylates and protects nuclear myosin 1c from proteasome-mediated degradation to activate rDNA transcription in early G1 AA Sarshad , M Corcoran, B Al-Muzzaini - PLoS , 2014 - journals.plos.orghttps://journals.plos.org/plosgenetics/article?id=10.1371/journal.pgen.1004390

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Our products are intended for lab use only. For any other use, please contact us.
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