Product correctly added to cart.

discount label
Mal-RTRPLWVRME-OH
View 3D

Biosynth logo

Mal-RTRPLWVRME-OH

Ref. 3D-PP46789

Undefined sizeTo inquire
Estimated delivery in United States, on Friday 25 Oct 2024

Product Information

Name:
Mal-RTRPLWVRME-OH
Synonyms:
  • Mal-Arg-Thr-Arg-Pro-Leu-Trp-Val-Arg-Met-Glu-OH
Description:

Peptide Mal-RTRPLWVRME-OH is a Research Peptide with significant interest within the field academic and medical research. This peptide is available for purchase at Cymit Quimica in multiple sizes and with a specification of your choice. Recent citations using Mal-RTRPLWVRME-OH include the following: Maternal antibiotic treatment protects offspring from diabetes development in nonobese diabetic mice by generation of tolerogenic APCs Y Hu, J Peng, N Tai, C Hu, X Zhang - The Journal of , 2015 - journals.aai.orghttps://journals.aai.org/jimmunol/article/195/9/4176/105082 Absolute quantitation of tryptophan-containing peptides and amyloid beta-peptide fragments by coulometric mass spectrometry Y Ai , P Zhao , PIJ Fnu , H Chen - of the American Society for Mass , 2021 - ACS Publicationshttps://pubs.acs.org/doi/abs/10.1021/jasms.1c00121 Development of Novel Protein Digestion and Quantitation Methods for Mass Spectrometric Analysis Y Ai - 2023 - search.proquest.comhttps://search.proquest.com/openview/198379ce38281219995295aa8e5e346a/1?pq-origsite=gscholar&cbl=18750&diss=y IRAK-M deficiency promotes the development of T1DM in NOD mice Q Tan, M Majewska-Szczepanik, X Zhang - Citeseerhttps://citeseerx.ist.psu.edu/document?repid=rep1&type=pdf&doi=a3a79874e1c2edec9b81a90714a81019bc6d2937 Paradoxical dampening of anti-islet self-reactivity but promotion of diabetes by OX40 ligand N Martin-Orozco , Z Chen, L Poirot, E Hyatt - The Journal of , 2003 - journals.aai.orghttps://journals.aai.org/jimmunol/article/171/12/6954/72063 Th17 cells promote pancreatic inflammation but only induce diabetes efficiently in lymphopenic hosts after conversion into Th1 cells N Martin-Orozco , Y Chung , SH Chang - European journal of , 2009 - Wiley Online Libraryhttps://onlinelibrary.wiley.com/doi/abs/10.1002/eji.200838475 CD28/CD154 blockade prevents autoimmune diabetes by inducing nondeletional tolerance after effector t-cell inhibition and regulatory T-cell expansion MR Rigby, AM Trexler, TC Pearson, CP Larsen - Diabetes, 2008 - Am Diabetes Assochttps://diabetesjournals.org/diabetes/article-abstract/57/10/2672/13402 MerTK regulates thymic selection of autoreactive T cells MA Wallet, RR Flores , Y Wang , Z Yi - Proceedings of the , 2009 - National Acad Scienceshttps://www.pnas.org/doi/abs/10.1073/pnas.0900683106 MerTK is required for apoptotic cell-induced T cell tolerance MA Wallet, P Sen , RR Flores , Y Wang , Z Yi - The Journal of , 2008 - rupress.orghttps://rupress.org/jem/article-abstract/205/1/219/47074 Anti-CD3 treatment upregulates PD-1 expression on activated effector T cells and severely impairs their inflammatory capacity M Wallberg, A Recino, J Phillips, D Howie - , 2017 - cyberleninka.orghttps://cyberleninka.org/article/n/142793.pdf Modulating the T Cell Response: Using Anti-Interleukin-7 Receptor-Alpha Monoclonal Antibodies with Autoantigen-Specific Immunotherapy to Prevent Type-1 M Lawson - 2019 - search.proquest.comhttps://search.proquest.com/openview/188a09eacf39f7949c7a53f2ba0c1a01/1?pq-origsite=gscholar&cbl=18750&diss=y TLR5-deficiency controls dendritic cell subset development in an autoimmune diabetes-susceptible model JA Pearson, Y Hu, J Peng, FS Wong - Frontiers in Immunology, 2024 - frontiersin.orghttps://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2024.1333967/full Inhibition of the proteasome reduces transfer-induced diabetes in nonobese diabetic mice J Petrovic , H Hall, R Mehr , R Glas - Scandinavian journal of , 2004 - Wiley Online Libraryhttps://onlinelibrary.wiley.com/doi/abs/10.1111/j.0300-9475.2004.01473.x Increased diabetes development and decreased function of CD4+CD25+ Treg in the absence of a functional DAP12 adaptor protein HTL Hall, H Sjölin, H Brauner - European journal of , 2008 - Wiley Online Libraryhttps://onlinelibrary.wiley.com/doi/abs/10.1002/eji.200838259 TRIF deficiency protects non-obese diabetic mice from type 1 diabetes by modulating the gut microbiota and dendritic cells E Gulden, C Chao, N Tai, JA Pearson, J Peng - Journal of , 2018 - Elsevierhttps://www.sciencedirect.com/science/article/pii/S0896841118302580 Peripherally induced regulatory T cells contribute to the control of autoimmune diabetes in the NOD mouse model C Schuster, F Jonas, F Zhao - European journal of , 2018 - Wiley Online Libraryhttps://onlinelibrary.wiley.com/doi/abs/10.1002/eji.201847498 Dendritic cell-targeted pancreatic beta-cell antigen leads to conversion of self-reactive CD4+ T cells into regulatory T cells and promotes immunotolerance in C Petzold, J Riewaldt, T Koenig - The review of diabetic , 2010 - ncbi.nlm.nih.govhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC2923380/ Foxp3+ Regulatory T Cells in Mouse Models of Type 1 Diabetes C Petzold, J Riewaldt, D Watts - Journal of diabetes , 2013 - Wiley Online Libraryhttps://onlinelibrary.wiley.com/doi/abs/10.1155/2013/940710

Notice:
Our products are intended for lab use only. For any other use, please contact us.
Brand:
Biosynth
Long term storage:
Notes:

Chemical properties

MDL:
Melting point:
Boiling point:
Flash point:
Density:
Concentration:
EINECS:
Merck:
HS code:

Hazard Info

UN Number:
EQ:
Class:
H Statements:
P Statements:
Forbidden to fly:
Hazard Info:
Packing Group:
LQ:

Technical inquiry about: 3D-PP46789 Mal-RTRPLWVRME-OH

Please use instead the cart to request a quotation or an order

If you want to request a quotation or place an order, please instead add the desired products to your cart and then request a quotation or order from the cart. It is faster, cheaper, and you will be able to benefit from the available discounts and other advantages.

* Mandatory fields.
Welcome to CymitQuimica!We use cookies to enhance your visit. We do not include advertising.

Please see our Cookies Policy for more details or adjust your preferences in "Settings".